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Molecular mechanisms of the hepatotocixity caused by acetaminophen

Where Untargeted Metabolomics Fills the MoA Gap Phenotypic hits without a target hypothesis Compounds identified through cell viability, phenotypic, or whole-organism screens often lack a clear molecular target

Se estudia por su capacidad para promover la reparacin tisular, la angiognesis y la cicatrizacin celular en mltiples sistemas orgnicos, incluyendo msculo, tendn, nervio y tejido gastrointestinal