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Allopurinol is crucial for promoting the secondary utilization of hypoxanthine and xanthine through a metabolic pathway intricately linked with hypoxanthine-guanine phosphoribosyl transferase

In some cases, alternative treatments with fewer metabolic effects might be appropriate, or the timing of therapies could be optimised to minimise conflicting effects whilst maintaining therapeutic benefit

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Theisen J (2000) Suppression of gastric acid secretion in patients with gastroesophageal reflux disease results in gastric bacterial overgrowth and deconjugation of bile acids

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