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Introduction Cell death is one of the fundamental characteristics and principles of all eukaryotic cells

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Excessive ROS levels can induce cellular damage, initiating a cascade of effects that potentially culminate in tissue and organ injury ( 3.1.1 Impact of ROS on cellular function and atherosclerosis Elevated ROS can induce endothelial cell dysfunction or death, disrupting the vascular barrier and leading to vasoconstrictive and vasodilatory dysfunction, vascular smooth muscle cell proliferation and migration, inflammatory responses, and thrombosisprocesses intimately associated with various cardiovascular disorders, including atherosclerosis, hypertension, and CHD (31)

Hence, complex formation between TBC1D9B and TMEM55B is mediated by multiple sites but does not involve the TBC domain that harbors its GAP activity

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It supports the immune system, helps the liver neutralize harmful substances, and protects DNA from damage