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The major effect of LTB4 and its receptor is to enhance the movement of leucocytes from the circulation toward the site of tissue damage (163)

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This was highlighted in a recent study performed in a mouse model of obesity-induced T2DM, in which an 8-week exendin-4 treatment, starting 3 days post-stroke, improved neurological recovery by reversing T2DM-induced atrophy of parvalbumin-positive GABAergic interneurons, which play a key role in neuroplasticity [8]