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In cell and animal studies, glucocorticoids reduce the gene expression that produces GLP-1 in intestinal L-cells, which is a clear and well-mapped mechanism

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There is also a hormonal argument

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Moreover, existing studies mostly focus on the regulation of RA synovial cells (such as FLS) or macrophages by the cGAS-STING pathway, whereas this study, for the first time, clearly demonstrates the central role of the cGAS-STING-ferroptosis axis in RA chondrocytes, and reveals that GLP-1RAs exerted therapeutic effects on RA through a dual mechanism of directly inhibiting this axis and simultaneously promoting the M2 polarization of macrophages