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Excessive deposition of extracellular matrix (ECM) and lung tissue remodelling is a central characteristic of fibrotic disorders and the ECM has been attributed a key role in the progressive nature of IPF 23

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Thrombin is the main activator and turns fibrinogen into fibrin

Three converging trends are driving adoption: (1) FDA shortage and supply constraints on brand-name GLP-1s increased demand for alternatives, (2) telemedicine platforms made compounded access seamless and affordable, and (3) genetic testing capabilities now allow personalization that brand-name formularies cannot match

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