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Mandal, Pravat K., et al

Researchers often use it in short courses targeted at specific infectious or dysbiotic contributions to autoimmune activation, rather than as a long-term immune-modulating agent

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We further compared the upregulated genes that were annotated as pathogen-responsive (based on WormCat 38 ) among the three sterile mutants and observed a significant overlap, where the changes in fem-3(-) represented a subset of those changed in glp-1(-) mutant (Supplementary Fig