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By activating its ubiquitously expressed receptor GLP1R, GLP-1 regulates multiple physiological processes which are summarized in Fig

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doi: 10.3390/microorganisms11030636 15 KroonSJRavelJHustonWM

S., Torres, D

This interaction may occur through two mechanisms: First, GLP-1 RA may decrease sodium reabsorption in the proximal tubule, leading to increased delivery of sodium chloride to the macula densa and subsequent activation of the tubuloglomerular feedback mechanism, which inhibits renin secretion [57]

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