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The activity and expression of NRF2 were decreased in many AKI and CKD animal models, suggesting that NRF2 was an important mediator in the pathogenesis and progression of AKI and CKD [204, 205]
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This mechanism represents a fundamentally different approach to fat metabolism compared to appetite suppression (GLP-1 agonists) or direct lipolysis stimulation (growth hormone peptides)
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serum valproic acid level 50-100 mg/L (350-700 uM/L): Therapeutic level
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