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Observational and preclinical data suggest that GLP-1 receptor activation may reduce neuroinflammation and slow the accumulation of amyloid and tau pathology associated with Alzheimer's disease, and semaglutide is currently in Phase 3 trials for both Alzheimer's disease and Parkinson's disease [12]

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Through direct suppression of T helper cell-mediated hyperinflammation and PKA-dependent modulation of local immune responses, GLP-1RAs help protect normal tissues (107)

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