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Upon the accumulation of misfolded proteins in the ER, the XBP1 mRNA undergoes specific splicing, generating the spliced XBP1 transcription factor (sXBP1), which subsequently translocates to the nucleus and drives the expression of stress-induced inflammatory genes

FOXO4-DRI disrupts this interaction and thus unlocks the door to apoptosis, the natural process of cell death

Individuals with excess methylation often do not do well with this form

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