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This suggests that its mechanism of action may be unconventional, possibly involving the modulation of multiple downstream pathways or direct interaction with cellular components like growth factor receptors or cytoskeletal proteins

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It also causes temporary (3 months) paralysis of the muscle being innervated by those nerve endings

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In preclinical models the fragment stimulates lipolysis, the breakdown of stored fat, and inhibits lipogenesis, the laying-down of new fat, with the proposed signal running through beta-3-adrenergic receptors on fat cells