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Exogenous MUFA administration, MUFA synthesis upregulation, and ACSL3-dependent MUFA membrane accumulation can all decrease the likelihood of ferroptosis [30, 33, 34]

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From day 1 of the experiment to day 5 the escape latency was found to remarkably decrease, however, escape latency in the APP/PS1 mice was higher than that of the wild type mice

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