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found human patients with congenital cerebellar ataxia with the same gain of function mutation in the GluR2 receptor as Lurcher mice, which also made them a human disease model [240]

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Elevates stress hormones

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In addition, the transcriptomic study revealed the activation of abscisic acid, auxin, jasmonic acid biosynthesis and signaling genes during GSH treatment (Cheng et al