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Glucagon receptor stimulation increases hepatic fatty acid oxidation and promotes lipolysis in adipose tissue through GIP receptor activation, creating metabolic conditions that favor fat utilization

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Each component addresses a different limitation of GLP-1 monotherapy, and the combination produces an effect profile that none of the three could achieve alone

GLP-1 receptor agonists as adjunct therapy in type 1 diabetes are gaining attention as clinicians look for new ways to improve glycemic control