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The chronic presence of biofilm-forming pathogens, combined with the sustained release of toxins and inflammatory mediators, creates an environment where the immune system is not able to resolve the infection effectively, leading to delayed wound healing, recurrent infections, and further tissue damage

found that GLP-1 receptor agonist use, compared with initiation of sodium-glucose cotransporter 2 (SGLT2) inhibitors, was associated with a lower risk of composite MALOs (aHR: 0.84, 95%CI: 0.74-0.94), including specifically lower occurrence of liver decompensation (aHR: 0.83) and all-cause mortality (aHR: 0.84) [56]