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Therefore, SCFAs deficiency may represent a contributing factor to PCOS symptomatology, potentially through multiple interconnected mechanisms including exacerbating ovarian inflammation, impairing follicular maturation, aggravating IR, and promoting follicular atresia via direct suppression of glucose uptake in granulosa cells
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Analysis of paired samples of GC tissue and adjacent normal mucosa revealed that the GC samples exhibited high ACTL6A and GCLC protein expression levels (Supplementary Fig
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